News and Views
Nature Medicine 13, 1413 - 1415 (2007)
doi:10.1038/nm1207-1413
Dying T cells trigger autoimmunity in HIV
Sarah Rowland-Jones1,2 & Tao Dong1
- Sarah Rowland-Jones and Tao Dong are in the Medical Research Council (MRC) Human Immunology Unit, Weatherall Institute of Molecular Medicine, John Radcliffe Hospital, Oxford OX3 9DS, UK.
- Sarah Rowland-Jones is also at the MRC Laboratories, PO Box 273, The Gambia, West Africa. e-mail: sarah.rowland-jones@ndm.ox.ac.uk
Abstract
During HIV-1 infection, there are many ways for CD4+ T cells to die. New findings suggest that an autoimmune mechanism may come into play (pages 1431–1439).
MORE ARTICLES LIKE THIS
These links to content published by NPG are automatically generated.
RESEARCH
Metalloproteases regulate T-cell proliferation and effector function via LAG-3The EMBO Journal Article (24 Jan 2007)
COMMD1 promotes the ubiquitination of NF-κB subunits through a cullin-containing ubiquitin ligaseThe EMBO Journal Article (24 Jan 2007)
Microbial translocation is a cause of systemic immune activation in chronic HIV infectionNature Medicine Article (01 Jan 2007)
Microbial translocation is a cause of systemic immune activation in chronic HIV infectionNature Medicine Article

