Nature Medicine
11, 952 - 958 (2005)
Published online: 4 September 2005; | doi:10.1038/nm1289
A crucial role for GRK2 in regulation of endothelial cell nitric oxide synthase function in portal hypertensionSongling Liu, Richard T Premont, Christopher D Kontos, Shoukang Zhu
& Don C Rockey Supplementary Fig. 1 (pdf 924K)
GRK5 and GRK6 expression in normal and injured sinusoidal endothelial cells. Supplementary Fig. 2 (pdf 2M)
Overexpression of GRK2 in sinusoidal endothelial cells does not affect the activation of the MAP kinase pathway. Supplementary Fig. 3 (pdf 1M)
Phosphorylation of GSK-3 by active Akt is decreased with increasing concentrations of GRK2. Supplementary Fig. 4 (pdf 972K)
Expression and purification of GRK2 fragments. Supplementary Fig. 5 (pdf 1M)
Agonist stimulation leads to Akt/GRK2 disassociation in injured sinusoidal endothelial cells. Supplementary Fig. 6 (pdf 2M)
GRK2 siRNA does not affect GRK5 and GRK6 expression. Supplementary Methods (pdf 100K)
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