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Nature Medicine  11, 409 - 417 (2005)
Published online: 27 March 2005; | doi:10.1038/nm1215

Calmodulin kinase II inhibition protects against structural heart disease

Rong Zhang, Michelle S C Khoo, Yuejin Wu, Yingbo Yang, Chad E Grueter, Gemin Ni, Edward E Price Jr., William Thiel, Silvia Guatimosim, Long-Sheng Song, Ernest C Madu, Anisha N Shah, Tatiana A Vishnivetskaya, James B Atkinson, Vsevolod V Gurevich, Guy Salama, W J Lederer, Roger J Colbran & Mark E Anderson
 
Figure 1 thumbnailFigure 1.
A genetic model of cardiac CaMKII inhibition.

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CaMKII inhibition improves adverse ventricular remodeling after myocardial infarction.

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Figure 3 thumbnailFigure 3.
CaMKII inhibition protects cellular mechanical function and preserves [Ca2+]i homeostasis after myocardial infarction.

Full FigureFull Figure and legend (174K)		 Figure 4 thumbnailFigure 4.
CaMKII is a downstream element in the betaAR signaling cascade.

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Figure 5 thumbnailFigure 5.
Cellular mechanical responses to betaAR stimulation are preserved during CaMKII inhibition.

Full FigureFull Figure and legend (130K)		 Figure 6 thumbnailFigure 6.
The effects of CaMKII inhibition on ICa.

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