1
Michael Platten is in the Department of Neurology, Department of General Neurology and Hertie-Institute for Clinical Brain Research, University of Tübingen, Tübingen, Germany.
2
Lawrence Steinman is in Department of Neurology and Neurological Sciences, Beckman Center for Molecular Medicine and the Interdepartmental Program in Immunology, Stanford University, Stanford, California, USA. steinman@stanford.edu
Two forms of glial cells have distinct roles in the neuroinflammatory cascade of a mouse model of multiple sclerosis. These cells serve as key targets for therapeutic intervention (pages 328−334 and 335−339).
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