Karl S Lang1, 7, Mike Recher1, 7, Tobias Junt1, Alexander A Navarini1, Nicola L Harris1, Stefan Freigang1, Bernhard Odermatt1, Curdin Conrad2, Lars M Ittner3, Stefan Bauer4, Sanjiv A Luther5, Satoshi Uematsu6, Shizuo Akira6, Hans Hengartner1
& Rolf M Zinkernagel11
Institute of Experimental Immunology, University Hospital of Zurich, Schmelzbergstrasse 12, Zurich, Switzerland.
2
Department of Dermatology, University Hospital of Zurich, Gloriastrasse 31, Zurich, Switzerland.
3
Research Laboratory for Calcium Metabolism, Clinic Balgrist, University of Zurich, Forchstr. 340, Zurich, Switzerland.
4
Institut für Medizinische Mikrobiologie, Immunologie und Hygiene, TU Munich, Trogerstr. 9, Munich, Germany.
5
Department of Biochemistry, University of Lausanne, Chemin des Boveresses 155, Epalinges, Switzerland.
6
Department of Host Defense, Osaka University, 3-1 Yamada-oka, Suita Osaka, Japan.
7
These authors contributed equally to this work.
Correspondence should be addressed to Karl S Lang karl.lang@usz.ch
production. These data identify the 'inflammatory status' of the target organ as a separate and limiting factor determining the development of autoimmune disease.
