The authors are at the Beatson Institute for Cancer Research, Glasgow G61 1BD, UK. K.Vousden@beatson.gla.ac.uk
Low levels of DNA damage caused by oxidative stress can be repaired, whereas extensive damage usually results in cell death. p53 contributes to both outcomes by stimulating expression of either pro- or antioxidant genes (pages 1306–1313).
MORE ARTICLES LIKE THIS
These links to content published by NPG are automatically generated.
