The author is in the Departments of Dermatology and Biochemistry and Molecular Biology, Mayo Clinic College of Medicine, Rochester, Minnesota 55905, USA. pittelkow.mark@mayo.edu
Psoriasis develops through interactions between the skin and immune system. The cell-signaling molecule Stat3 now emerges as a bridge between the two, initiating perturbations in the epidermis and generating misguided T-cell responses (pages 43−49).
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