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Article
Nature Medicine  10, 696 - 703 (2004)
Published online: 6 June 2004; | doi:10.1038/nm1059

LARGE can functionally bypass alpha-dystroglycan glycosylation defects in distinct congenital muscular dystrophies

Rita Barresi, Daniel E Michele, Motoi Kanagawa, Hollie A Harper, Sherri A Dovico, Jakob S Satz, Steven A Moore, Wenli Zhang, Harry Schachter, Jan P Dumanski, Ronald D Cohn, Ichizo Nishino & Kevin P Campbell

Supplementary Fig. 1 (pdf 176K)
DGC expression in Largemyd skeletal muscle after LARGE gene transfer.

Supplementary Fig. 2 (pdf 210K)
Improved morphology and function of Largemyd transduced skeletal muscle.

Supplementary Fig. 3 (pdf 284K)
Forced expression of POMGnT1 does not improve the functional glycosylation of alpha-DG.


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