Fred S Harman1, 2, 5, Christopher J Nicol3, 5, Holly E Marin1, 2, Jerrold M Ward4, Frank J Gonzalez3
& Jeffrey M Peters1, 21
Department of Veterinary Science and The Center for Molecular Toxicology and Carcinogenesis, The Pennsylvania State University, University Park, Pennsylvania
16802, USA.
2
Graduate Program in Biochemistry, Microbiology, and Molecular Biology, The Pennsylvania State University, University Park, Pennsylvania
16802, USA.
3
Laboratory of Metabolism, National Cancer Institute, Bethesda, Maryland
20892, USA.
4
Veterinary and Tumor Pathology Section, Office of Laboratory Animal Resources, National Cancer Institute, Frederick, Maryland
21702, USA.
5
These authors contributed equally to this work.
Correspondence should be addressed to Jeffrey M Peters jmp21@psu.edu
attenuates colon carcinogenesis
(PPAR-
) is expressed at high levels in colon tumors, but its contribution to colon cancer is unclear. We examined the role of PPAR-
