Haibin Wang1, Yong Guo1, Dingzhi Wang2, Philip J Kingsley3, Lawrence J Marnett3, Sanjoy K Das1, 4, Raymond N DuBois2, 4, 5
& Sudhansu K Dey1, 5, 61
Department of Pediatrics, Division of Reproductive and Developmental Biology, Vanderbilt University Medical Center, 1161 21st Avenue South, Nashville, Tennessee 37232, USA.
2
Department of Medicine, Division of Reproductive and Developmental Biology, Vanderbilt University Medical Center, 1161 21st Avenue South, Nashville, Tennessee 37232, USA.
3
Department of Biochemistry, Institute of Chemical Biology, Division of Reproductive and Developmental Biology, Vanderbilt University Medical Center, 1161 21st Avenue South, Nashville, Tennessee 37232, USA.
4
Department of Cancer Biology, Division of Reproductive and Developmental Biology, Vanderbilt University Medical Center, 1161 21st Avenue South, Nashville, Tennessee 37232, USA.
5
Department of Pharmacology, Division of Reproductive and Developmental Biology, Vanderbilt University Medical Center, 1161 21st Avenue South, Nashville, Tennessee 37232, USA.
6
Departments of Cell and Developmental Biology, Division of Reproductive and Developmental Biology, Vanderbilt University Medical Center, 1161 21st Avenue South, Nashville, Tennessee 37232, USA.
Correspondence should be addressed to Sudhansu K Dey sk.dey@vanderbilt.edu
-adrenergic receptor agonist. Impaired oviductal embryo transport is also observed in wild-type mice treated with methanandamide. Collectively, the results suggest that aberrant cannabinoid signaling impedes coordinated oviductal smooth muscle contraction and relaxation crucial to normal oviductal embryo transport. Colocalization of CB1 and 
