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Letter
Nature Genetics 34, 102–107 (1 May 2003) | doi:10.1038/ng1152
Regulatory defects in liver and intestine implicate abnormal hepcidin and Cybrd1 expression in mouse hemochromatosis
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Abstract
Individuals with hereditary hemochromatosis suffer from systemic iron overload due to duodenal hyperabsorption. Most cases arise from a founder mutation in HFE (845G|[rarr]|A; ref.
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