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Letter
Nature Genetics 10, 111–113 (1 May 1995) | doi:10.1038/ng0595-111
A candidate genetic risk factor for vascular disease: a common mutation in methylenetetrahydrofolate reductase
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Abstract
Hyperhomocysteinaemia has been identified as a risk factor for cerebrovascular, peripheral vascular and coronary heart disease1–4. Elevated levels of plasma homocysteine can result from genetic or nutrient-related disturbances in the trans-sulphuration or re-methylation pathways for homocysteine metabolism1,5–7.
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