Distinct mutations of FLT3 result in different outcomes in patients with AML
This article has no abstract so we have provided the first paragraph of the full text.
In patients with acute myeloid leukemia (AML), internal tandem duplications (ITDs) within the tyrosine kinase protein FLT3 correlate with increased relapse rates and poor overall survival (OS); however, the prognostic impact of mutations in the tyrosine kinase domain (TKD) of FLT3 is currently unknown.
Full text of this article is available with one of the following:
- Personal subscription Purchase your own personal subscription to this journal. Already a subscriber? Please log in for immediate access.
- 7 day single article pass for US$18 In order to purchase this article you must be a registered user. Please register or log in.
- Site licence Learn more about institutional site licences
Current Subscribers
Please log in to access the full text article using the login box at the top of the page.
MORE ARTICLES LIKE THIS
These links to content published by NPG are automatically generated.