Inhibition of specific brain functions might treat post-traumatic stress disorder
This article has no abstract so we have provided the first paragraph of the full text.
Post-traumatic stress disorder (PTSD) has been associated with reduced ventromedial prefrontal cortex (vmPFC) volumes and overactivation of the amygdala, but it is unclear whether these features reflect the cause or a secondary effect of this disorder. Koenigs et al. report on phase III of the Vietnam Head Injury Study, in which the association of brain damage with PTSD was investigated.
Full text of this article is available with one of the following:
- Personal subscription Purchase your own personal subscription to this journal. Already a subscriber? Please log in for immediate access.
- 7 day single article pass for US$18 In order to purchase this article you must be a registered user. Please register or log in.
- Site licence Learn more about institutional site licences
Current Subscribers
Please log in to access the full text article using the login box at the top of the page.