Targeting T cells to prevent secondary damage following traumatic brain injury
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Nitrone radical scavengers have been shown to improve morphological and functional outcome in animal models of traumatic brain injury (TBI). Clausen et al. hypothesized that nonpenetrating nitrones act systemically by interacting with the peripheral immune system at the microvascular level to prevent the secondary injury cascade following TBI. To test this theory, they examined whether 2-sulfophenyl-N-tert-butyl-nitrone (S-PBN) modulated the trafficking of damaging components of the immune response in a rat model of TBI.
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