Article abstract


Nature Chemical Biology 5, 421 - 427 (2009)
Published online: 3 May 2009 | doi:10.1038/nchembio.168

In vivo effects of a GPR30 antagonist

Megan K Dennis1, Ritwik Burai2, Chinnasamy Ramesh2, Whitney K Petrie1, Sara N Alcon1, Tapan K Nayak1, Cristian G Bologa3, Andrei Leitao3, Eugen Brailoiu4, Elena Deliu4, Nae J Dun4, Larry A Sklar5,6, Helen J Hathaway1,5, Jeffrey B Arterburn2,5, Tudor I Oprea3,5 & Eric R Prossnitz1,5


Estrogen is central to many physiological processes throughout the human body. We have previously shown that the G protein–coupled receptor GPR30 (also known as GPER), in addition to classical nuclear estrogen receptors (ERalpha and ERbeta), activates cellular signaling pathways in response to estrogen. In order to distinguish between the actions of classical estrogen receptors and GPR30, we have previously characterized G-1 (1), a selective agonist of GPR30. To complement the pharmacological properties of G-1, we sought to identify an antagonist of GPR30 that displays similar selectivity against the classical estrogen receptors. Here we describe the identification and characterization of G15 (2), a G-1 analog that binds to GPR30 with high affinity and acts as an antagonist of estrogen signaling through GPR30. In vivo administration of G15 revealed that GPR30 contributes to both uterine and neurological responses initiated by estrogen. The identification of this antagonist will accelerate the evaluation of the roles of GPR30 in human physiology.

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  1. Department of Cell Biology & Physiology, University of New Mexico Health Sciences Center, Albuquerque, New Mexico, USA.
  2. Department of Chemistry and Biochemistry, New Mexico State University, Las Cruces, New Mexico, USA.
  3. Division of Biocomputing, Department of Biochemistry & Molecular Biology, University of New Mexico Health Sciences Center, Albuquerque, New Mexico, USA.
  4. Department of Pharmacology, Temple University School of Medicine, Philadelphia, Pennsylvania, USA.
  5. University of New Mexico Cancer Center, University of New Mexico Health Sciences Center, Albuquerque, New Mexico, USA
  6. Department of Pathology, University of New Mexico Health Sciences Center, Albuquerque, New Mexico, USA.

Correspondence to: Eric R Prossnitz1,5 e-mail: eprossnitz@salud.unm.edu

Correspondence to: Tudor I Oprea3,5 e-mail: toprea@salud.unm.edu

Correspondence to: Jeffrey B Arterburn2,5 e-mail: jarterbu@nmsu.edu



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