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Letter
Nature Cell Biology 9, 1303–1310 (1 November 2007) | doi:10.1038/ncb1650
AK2 activates a novel apoptotic pathway through formation of a complex with FADD and caspase-10
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Abstract
Mitochondrial proteins function as essential regulators in apoptosis. Here, we show that mitochondrial adenylate kinase 2 (AK2) mediates mitochondrial apoptosis through the formation of an AK2–FADD–caspase-10 (AFAC10) complex.
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