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Brief Communication
Nature Cell Biology 2, 376–378 (1 June 2000) | doi:10.1038/35014075
Latrunculin alters the actin-monomer subunit interface to prevent polymerization
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Abstract
Latrunculin-A is a drug that is capable of rapidly, reversibly and specifically disrupting the actin cytoskeleton. The efficacy of its action has made it a compound of choice in many cell-biology laboratories, supplanting the classic actin-depolymerizing drug cytochalasin-D.
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