Access
To read this article in full you may need to log in, make a payment or gain access through a site license (see right).
Letter
Nature Cell Biology 10, 361–369 (1 March 2008) | doi:10.1038/ncb1699
VHL loss actuates a HIF-independent senescence programme mediated by Rb and p400
&
Abstract
Germline von Hippel–Lindau tumour suppressor gene (VHL) mutations cause renal cell carcinomas, haemangioblastomas and phaeochromocytomas in humans. Mutations in VHL also occur in sporadic renal cell carcinomas.
To read this article in full you may need to log in, make a payment or gain access through a site license (see right).
