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CDK targets Sae2 to control DNA-end resection and homologous recombination

Pablo Huertas, Felipe Cortés-Ledesma, Alessandro A. Sartori, Andrés Aguilera & Stephen P. Jackson

Nature 455, 689-692(2 October 2008)

doi:10.1038/nature07215

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Figure 1 - Unfortunately we are unable to provide accessible alternative text for this. If you require assistance to access this image, or to obtain a text description, please contact npg@nature.com

Figure 1

Ser 267 mutation impairs Sae2 function.

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Figure 2

Sae2 is phosphorylated by Cdc28 on Ser 267.

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Figure 3

DNA-end resection is controlled by Sae2.

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Figure 4

Sae2 mutations affect Mre11 and Rad52 dynamics, and DSB repair.

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