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From the following article:

A phosphatase complex that dephosphorylates gammaH2AX regulates DNA damage checkpoint recovery

Michael-Christopher Keogh, Jung-Ae Kim, Michael Downey, Jeffrey Fillingham, Dipanjan Chowdhury, Jacob C. Harrison, Megumi Onishi, Nira Datta, Sarah Galicia, Andrew Emili, Judy Lieberman, Xuetong Shen, Stephen Buratowski, James E. Haber, Daniel Durocher, Jack F. Greenblatt & Nevan J. Krogan

Nature 439, 497-501(26 January 2006)

doi:10.1038/nature04384

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Figure 1 - Unfortunately we are unable to provide accessible alternative text for this. If you require assistance to access this image, or to obtain a text description, please contact npg@nature.com

Figure 1

The HTP-C regulates H2A in vivo and dephosphorylates bold gammaH2AX in vitro.

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Figure 2

The HTP-C interacts genetically with DNA repair factors.

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Figure 3

The HTP-C is not required for DSB repair and dephosphorylates displaced bold gammaH2AX rather than chromatin-associated bold gammaH2AX at a DSB.

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Figure 4

bold gammaH2AX dephosphorylation by the HTP-C is required for DNA damage checkpoint recovery.

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