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Mal (MyD88-adapter-like) is required for Toll-like receptor-4 signal transduction

Katherine A. Fitzgerald, Eva M. Palsson-McDermott, Andrew G. Bowie, Caroline A. Jefferies, Ashley S. Mansell, Gareth Brady, Elizabeth Brint, Aisling Dunne, Pearl Gray, Mary T. Harte, Diane McMurray, Dirk E. Smith, John E. Sims, Timothy A. Bird and Luke A. J. O'Neill

Nature 413, 78-83(6 September 2001)

doi:10.1038/35092578

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Figure 1 - Unfortunately we are unable to provide accessible alternative text for this. If you require assistance to access this image, or to obtain a text description, please contact npg@nature.com

Figure 1

MyD88-adapter-like (Mal) is a widely expressed homologue of MyD88.

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Figure 2

Mal overexpression activated NF-kappaB, JNK and ERK1/2.

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Figure 3

Mal signals by means of MyD88, IRAK-2, TRAF-6 and TAK.

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Figure 4

Mal is required for TLR-4 and LPS signalling.

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