Kidney International (1984) 25, 857–868; doi:10.1038/ki.1984.102
Contribution of the renin-angiotensin system to the control of intrarenal hemodynamics
L Gabriel Navar1 and L
szl
Rosivall1
1Department of Physiology and Biophysics, Nephrology Research and Training Center, University of Alabama in Birmingham Medical Center, Birmingham, Alabama
Correspondence: Dr L G Navar, Department of Physiology and Biophysics, Nephrology Research and Training Center, University of Alabama in Birmingham, University Station, 727 CDLD Building, Birmingham, Alabama 35294, USA
Received 16 March 1983; Revised 17 August 1983.
Top of pageAbstract
Intensive investigative efforts probing the many characteristics and potential functions of the renin-angiotensin system (RAS) have resulted in an exponentially expanding literature. In this editorial, we focus on the renal hemodynamic influences exerted by the RAS as a local regulatory mechanism. Indeed, local control was one initial function ascribed to the RAS by early investigators in this area. Because of its unique structure, the juxtaglomerular apparatus, now known to be the major site of renin formation, intrigued morphologists. It was postulated that the cells of the juxtaglomerular apparatus could control blood flow through the glomerulus by swelling and shrinking the afferent arteriolar lumen [1]. The unique juxtaposition of the macula densa cells of the early distal tubule to the glomerular arterioles also suggested mechanisms linking the function of the tubules with that of the arterioles [2–4]. Goormaghtigh [5] proposed an endocrine function for the juxtaglomerular apparatus and demonstrated uncanny insight in postulating the release of an agent by this structure that could activate or form a vasoactive substance which, in turn, influenced local vascular tone. Although some investigators continued to support the notion that local control was a major function of the RAS [6–8], the major areas of investigation in this field were oriented toward non-renal functions of the system such as in the control of aldosterone release, the regulation of systemic cardiovascular function, and the pathophysiology of hypertension [9]. Recent investigations, however, have renewed enthusiasm regarding the possibility that locally formed angiotensin II (All) may participate as an important modulator of renal vascular tone.
Top of pageReferences
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