Original Article
Journal of Perinatology (2009) 29, 698–701; doi:10.1038/jp.2009.69; published online 25 June 2009
Methemoglobin to cumulative nitric oxide ratio and response to inhaled nitric oxide in PPHN
M J Pabalan1, S P Nayak1, R M Ryan1, V H Kumar1 and S Lakshminrusimha1
1Department of Pediatrics, Division of Neonatology, University at Buffalo, Women and Children's Hospital of Buffalo, Buffalo, NY, USA
Correspondence: Dr S Lakshminrusimha, Department of Pediatrics, Division of Neonatology, University of Buffalo, Women and Children's Hospital of Buffalo, 219 Bryant Street, Buffalo, NY 14222, USA. E-mail: slakshmi@buffalo.edu
Received 24 August 2008; Revised 21 January 2009; Accepted 25 February 2009; Published online 25 June 2009.
Abstract
Background:
One-third of infants with persistent pulmonary hypertension of the newborn (PPHN) do not respond to inhaled nitric oxide (iNO). If iNO is not delivered to the pulmonary vasculature because of parenchymal lung disease, it cannot interact with hemoglobin to form methemoglobin (MHb).
Objective:
To study the correlation between oxygenation response to iNO in infants with PPHN secondary to parenchymal lung disease and initial MHb% to cumulative NO exposure (ppm
hours) ratio (MHb/
NO).
Study Design:
Retrospective chart review of neonates with PPHN secondary to parenchymal lung disease treated with iNO comparing non-responders (PaO2/FiO2 ratio<10 change with iNO) with responders (
10 change).
Result:
Non-responders (n=16) had a PaO2/FiO2 of 83
48 (mean
s.d.) and decreased to 74
44 after iNO. PaO2/FiO2 increased from 70
48 to 151
63 with iNO among responders (n=36). The MHb/
NO ratio was low (0.024
0.012) among non-responders compared with responders (0.07
0.053, P<0.005).
Conclusion:
Inadequate oxygenation response to iNO is associated with lower MHb/
NO, suggesting suboptimal delivery of iNO to the pulmonary vasculature.
Keywords:
lung recruitment, oxygenation, pulmonary hypertension, hypoxia
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