Mathias Köppen1, Jeffrey S. Simske2, Paul A. Sims2, Bonnie L. Firestein3, David H. Hall4, Anthony D. Radice5, Christopher Rongo6
& Jeffrey D. Hardin1, 21
Program in Cellular and Molecular Biology, University of Wisconsin-Madison, 1117 West Johnson Street, Madison, Wisconsin 53706, USA
2
Department of Zoology, University of Wisconsin-Madison, 1117 West Johnson Street, Madison, Wisconsin 53706, USA
3
Department of Cell Biology and Neuroscience, Rutgers University, Piscataway, New Jersey 08854, USA
4
Center for C. elegans Anatomy, Department of Neuroscience, Albert Einstein College of Medicine, 1410 Pelham Parkway, Bronx, New York 10461, USA
5
Department of Stem Cell Biology, Lindsley F. Kimball Research Institute, New York Blood Center, 310 East 67th Street, New York, New York 10021, USA
6
Department of Genetics, The Waksman Institute, Rutgers University, Piscataway, New Jersey 08854, USA
Correspondence should be addressed to Jeffrey D. Hardin jdhardin@facstaff.wisc.eduPublished 8 October 2001
-catenin) localization is only mildly affected. We conclude that LET-413 and DLG-1 cooperatively control AJM-1 localization and that AJM-1 controls the integrity of a distinct apical junctional domain in C. elegans.
