Nature Medicine homepage
Advanced search
 Journal home > Archive > Table of Contents > News and Views > Abstract
Journal home
Advance online publication
Current issue
Archive
Press releases
Supplements
Focuses
Guide to authors
Online submissionOnline submission
For referees
Free online issue
Contact the journal
Subscribe
Advertising
work@npg
Reprints and permissions
About this site
For librarians
 
NPG Resources
Nature
Nature Reviews
Nature Immunology
Nature Cell Biology
Nature Genetics
news@nature.com
Nature Conferences
Dissect Medicine
NPG Subject areas
Biotechnology
Cancer
Chemistry
Clinical Medicine
Dentistry
Development
Drug Discovery
Earth Sciences
Evolution & Ecology
Genetics
Immunology
Materials Science
Medical Research
Microbiology
Molecular Cell Biology
Neuroscience
Pharmacology
Physics
Browse all publications
News and Views
Nature Medicine  5, 1346 - 1347 (1999)
doi:10.1038/70907

Is it thee or me?—autoimmunity in Lyme disease

Mark S. Klempner1 & Brigitte T. Huber2

1  Department of Molecular Biology and Microbiology Tufts University School of Medicine 750 Washington Street Boston, Massachusetts 02111,USA

2  Department of Pathology Tufts University School of Medicine Boston, Massachusetts 02111, USA
mark.klempner@es.nemc.org

The chronic musculskeletal and nervous system symptoms that persist after Lyme disease infection are believed to be caused by an autoimmune reaction triggered by B. burgdorferi. A new technique should allow us to identify the self-reactive T cells and antigen epitopes involved (1375−1382).

 Top
Abstract
Previous | Next
Table of contents
Full textFull text
Download PDFDownload PDF
Send to a friendSend to a friend
Save this linkSave this link
Figures & Tables
See also: Article by Hemmer et al.
Export citation
natureproducts

Search buyers guide:

 
ADVERTISEMENT
 
Nature Medicine
ISSN: 1078-8956
EISSN: 1546-170X		 Journal home | Advance online publication | Current issue | Archive | Press releases | Supplements | Focuses | For authors | Online submission | For referees | Free online issue | About the journal | Contact the journal | Subscribe | Advertising | work@npg | Reprints and permissions | About this site | For librarians
Nature Publishing Group, publisher of Nature, and other science journals and reference works©1999 Nature Publishing Group | Privacy policy