Correction to: Oncogene (2009) 28, 3563–3572; doi:10.1038/onc.2009.213; published online 20 July 2009
Owing to a typesetting error, Figure 1a was published incorrectly. The correct version of the figure is given here.
Requirement of VDAC2 for apoptosis in Bak-silenced cells. (a) Wild-type (WT) and ΔVDAC2 cells were silenced with the indicated siRNAs. The control was non-targeting siRNA. Then cell lysates were subjected to immunoblotting with antibodies for Bak, Bax, VDAC2 and GAPDH (GAPDH was the loading control). (b–j) Suppression of apoptosis in Bak-silenced ΔVDAC2 cells. WT (crosses) and ΔVDAC2 (closed circles) cells were silenced with siRNAs for the non-targeting control (b, e and h), Bax (c, f and i) or Bak (d, g and j), and then were incubated with 2 μg/ml of tunicamycin (b–d), 40 μM etoposide (e–g) or 5 μM thapsigargin (h–j) for the indicated periods. Cell viability was assessed by propidium iodide (PI) staining. Data are shown as the mean±s.d. (n=3, *P<0.05 versus WT cells).
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The online version of the original article can be found at 10.1038/onc.2009.213
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Yamagata, H., Shimizu, S., Nishida, Y. et al. Erratum: Requirement of voltage-dependent anion channel 2 for pro-apoptotic activity of Bax. Oncogene 29, 2892 (2010). https://doi.org/10.1038/onc.2010.98
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DOI: https://doi.org/10.1038/onc.2010.98
