Abstract
WE have presented evidence which suggests that a high salt intake may be related to the development of human essential hypertension1, and that a prolonged low salt intake is compatible with apparently normal adrenocortical function2.
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References
a, Dahl, L. K., Nature, 181, 989 (1958); b, Dahl, L. K., in Essential Hypertension, An International Symposium, edit. by Cottier, P., and Bock, K. D., Berne, 53 (Springer-Verlag, Heidelberg, 1960).
Dahl, L. K., Silver, L., Christie, R. W., and Genest, J., Nature, 185, 110 (1960).
Dahl, L. K., New England J. Med., 258, 1152, 1205 (1958).
Jenkins, D., Forsham, P. H., Laidlaw, J. C., Reddy, W. J., and Thorn, G. W., Amer. J. Med., 18, 3 (1955).
Dahl, L. K., J. Clin. Nutr., 6, 1 (1958).
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DAHL, L., SILVER, L., SPRARAGEN, S. et al. Effect of Chronic High and Low Salt Intakes on Adrenocortical Function. Nature 190, 348–349 (1961). https://doi.org/10.1038/190348a0
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DOI: https://doi.org/10.1038/190348a0
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