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Letter |
Metabolic reprogramming by the S-nitroso-CoA reductase system protects against kidney injury
AKR1A1-regulated protein S-nitrosylation protects against kidney injury through PKM2-mediated metabolic reprogramming.
- Hua-Lin Zhou
- , Rongli Zhang
- & Jonathan S. Stamler
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Letter |
Molecular modifiers reveal a mechanism of pathological crystal growth inhibition
Like citrate, the molecule hydroxycitrate is shown to inhibit growth of the crystal that is the principal component of kidney stones, suggesting that hydroxycitrate could be another treatment for kidney stone disease.
- Jihae Chung
- , Ignacio Granja
- & Jeffrey D. Rimer
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News |
The hidden threat of West Nile virus
Researchers probe possible link with kidney disease.
- Amy Maxmen
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Letter |
Mutations in kelch-like 3 and cullin 3 cause hypertension and electrolyte abnormalities
Exome sequencing identifies mutations in kelch-like 3 and cullin 3 as causes of a syndrome featuring high blood pressure and electrolyte abnormalities.
- Lynn M. Boyden
- , Murim Choi
- & Richard P. Lifton
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News |
Biomarkers for kidney damage should speed drug development
Drug-safety indicators are the first fruits of a collaboration between academia, industry and regulators.
- Brendan Borrell
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- Acid, base, fluid, electrolyte disorders
- Acute kidney injury
- Chronic kidney disease
- Fanconi syndrome
- Glomerular diseases
- Interstitial disease
- Lupus nephritis
- Nephritis
- Nephrosclerosis
- Obstructive nephropathy
- Paediatric kidney disease
- Phosphorus metabolism disorders
- Polycystic kidney disease
- Renal artery stenosis
- Renal calculi
- Renal cancer
- Renal fibrosis
- Thrombotic microangiopathies
- Toxin-induced nephropathy
- Urinary tract infection